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rabbit anti mouse tgf β neutralizing mab  (R&D Systems)


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    Structured Review

    R&D Systems rabbit anti mouse tgf β neutralizing mab
    Fig. 3 AHR and inflammatory features of naive HFD and OVA-HFD models. AHR to MCh (a), inflammatory cell profile in the BALF (b), IL-4 (c) and IL-5 levels (d), and <t>TGF-β1</t> (e) concentrations in lung homogenates. Immunohistochemical staining for TGF-β1 (F) in tissue from naïve HFD, OVA-HFD, naive ND, and OVA-ND mice are shown. †P < 0.05; ‡P < 0.01; *P < 0.001. AHR airway hyperresponsiveness, MCh methacholine, BALF bronchoalveolar lavage fluid, HFD high-fat-diet, IL interleukin, OVA ovalbumin, ND normal diet, TGF transforming growth factor
    Rabbit Anti Mouse Tgf β Neutralizing Mab, supplied by R&D Systems, used in various techniques. Bioz Stars score: 94/100, based on 118 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/rabbit anti mouse tgf β neutralizing mab/product/R&D Systems
    Average 94 stars, based on 118 article reviews
    rabbit anti mouse tgf β neutralizing mab - by Bioz Stars, 2026-06
    94/100 stars

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    1) Product Images from "Insulin resistance mediates high-fat diet-induced pulmonary fibrosis and airway hyperresponsiveness through the TGF-β1 pathway."

    Article Title: Insulin resistance mediates high-fat diet-induced pulmonary fibrosis and airway hyperresponsiveness through the TGF-β1 pathway.

    Journal: Experimental & molecular medicine

    doi: 10.1038/s12276-019-0258-7

    Fig. 3 AHR and inflammatory features of naive HFD and OVA-HFD models. AHR to MCh (a), inflammatory cell profile in the BALF (b), IL-4 (c) and IL-5 levels (d), and TGF-β1 (e) concentrations in lung homogenates. Immunohistochemical staining for TGF-β1 (F) in tissue from naïve HFD, OVA-HFD, naive ND, and OVA-ND mice are shown. †P < 0.05; ‡P < 0.01; *P < 0.001. AHR airway hyperresponsiveness, MCh methacholine, BALF bronchoalveolar lavage fluid, HFD high-fat-diet, IL interleukin, OVA ovalbumin, ND normal diet, TGF transforming growth factor
    Figure Legend Snippet: Fig. 3 AHR and inflammatory features of naive HFD and OVA-HFD models. AHR to MCh (a), inflammatory cell profile in the BALF (b), IL-4 (c) and IL-5 levels (d), and TGF-β1 (e) concentrations in lung homogenates. Immunohistochemical staining for TGF-β1 (F) in tissue from naïve HFD, OVA-HFD, naive ND, and OVA-ND mice are shown. †P < 0.05; ‡P < 0.01; *P < 0.001. AHR airway hyperresponsiveness, MCh methacholine, BALF bronchoalveolar lavage fluid, HFD high-fat-diet, IL interleukin, OVA ovalbumin, ND normal diet, TGF transforming growth factor

    Techniques Used: Immunohistochemical staining, Staining



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    94
    R&D Systems rabbit anti mouse tgf β neutralizing mab
    Fig. 3 AHR and inflammatory features of naive HFD and OVA-HFD models. AHR to MCh (a), inflammatory cell profile in the BALF (b), IL-4 (c) and IL-5 levels (d), and <t>TGF-β1</t> (e) concentrations in lung homogenates. Immunohistochemical staining for TGF-β1 (F) in tissue from naïve HFD, OVA-HFD, naive ND, and OVA-ND mice are shown. †P < 0.05; ‡P < 0.01; *P < 0.001. AHR airway hyperresponsiveness, MCh methacholine, BALF bronchoalveolar lavage fluid, HFD high-fat-diet, IL interleukin, OVA ovalbumin, ND normal diet, TGF transforming growth factor
    Rabbit Anti Mouse Tgf β Neutralizing Mab, supplied by R&D Systems, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/rabbit anti mouse tgf β neutralizing mab/product/R&D Systems
    Average 94 stars, based on 1 article reviews
    rabbit anti mouse tgf β neutralizing mab - by Bioz Stars, 2026-06
    94/100 stars
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    Fig. 3 AHR and inflammatory features of naive HFD and OVA-HFD models. AHR to MCh (a), inflammatory cell profile in the BALF (b), IL-4 (c) and IL-5 levels (d), and TGF-β1 (e) concentrations in lung homogenates. Immunohistochemical staining for TGF-β1 (F) in tissue from naïve HFD, OVA-HFD, naive ND, and OVA-ND mice are shown. †P < 0.05; ‡P < 0.01; *P < 0.001. AHR airway hyperresponsiveness, MCh methacholine, BALF bronchoalveolar lavage fluid, HFD high-fat-diet, IL interleukin, OVA ovalbumin, ND normal diet, TGF transforming growth factor

    Journal: Experimental & molecular medicine

    Article Title: Insulin resistance mediates high-fat diet-induced pulmonary fibrosis and airway hyperresponsiveness through the TGF-β1 pathway.

    doi: 10.1038/s12276-019-0258-7

    Figure Lengend Snippet: Fig. 3 AHR and inflammatory features of naive HFD and OVA-HFD models. AHR to MCh (a), inflammatory cell profile in the BALF (b), IL-4 (c) and IL-5 levels (d), and TGF-β1 (e) concentrations in lung homogenates. Immunohistochemical staining for TGF-β1 (F) in tissue from naïve HFD, OVA-HFD, naive ND, and OVA-ND mice are shown. †P < 0.05; ‡P < 0.01; *P < 0.001. AHR airway hyperresponsiveness, MCh methacholine, BALF bronchoalveolar lavage fluid, HFD high-fat-diet, IL interleukin, OVA ovalbumin, ND normal diet, TGF transforming growth factor

    Article Snippet: To block TGF-β1, a rabbit anti-mouse TGF-β neutralizing mAb (100 μg/mouse; R&D Systems Inc., Minneapolis, MN, USA) or rabbit IgG antibody (100 μg/ mouse; R&D Systems) was administered once via the intravenous (i.v.) route 4 h before the first OVA challenge (Fig. 1).

    Techniques: Immunohistochemical staining, Staining